Selected publications

  • Grossi, E; Huarte, M. (2018) Non-coding RNA: A lncRNA Guarding genome integrity. Nature Cell Biology. 20(4):371-372.

  • Marchese, FP; Huarte, M. (2018) A "Counter-Enhancer'' in Tumor Suppression. Cell. 173(6):1318-1319.

  • Marchese FP, Raimondi I, Huarte M. (2017) The multidimensional mechanisms of long noncoding RNA function. Genome Biol. Oct 31;18(1):206

  • Marín-Béjar O, Mas AM, González J, Martinez D, Athie A, Morales X, Galduroz M, Raimondi I, Grossi E, Guo S, Rouzaut A, Ulitsky I, Huarte M. (2017) The human lncRNA LINC-PINT inhibits tumor cell invasion through a highly conserved sequence element. Genome Biol. Oct 27;18(1):202

  • Marchese FP, Grossi E, Marín-Béjar O, Bharti SK, Raimondi I, González J, Martínez-Herrera DJ, Athie A, Amadoz A, Brosh RM and Huarte M (2016). A lncRNA regulates sister chromatid cohesion. Mol Cell. Aug 4;63(3):397-407.

  • Huarte M (2015) The emerging roles of lncRNAs in cancer. Nat Med. 2015 Nov;21(11):1253-61

  • Sánchez Y, Segura V, Marín-Béjar O, Athie A, Marchese FP, González J, Bujanda L, Guo S, Matheu A and Huarte M (2014). Genome-wide analysis of the human p53 transcriptional network unveils a lncRNA tumor suppressor signature. Nature Communications, 5:5812.

  • Marin-Bejar O, Marchese FP, Athie A, Sanchez Y, Gonzalez J, Segura V, Huang L, Moreno I, Navarro A, Monzo M, Garcia-Foncillas J, Rinn JL, Guo S, Huarte M (2013). Pint lincRNA connects the p53 pathway with epigenetic silencing by the Polycomb repressive complex 2. Genome Biol 14, R104.

  • Huarte, M*, Guttman, M., Feldser, D., Garber, M., Khalil, A.M., Zuk, O., Amit, I., Regev, A., Lander, E.S., Jacks, T., Rinn, J.L*. (2010). A large intergenic noncoding RNA induced by p53 mediates global gene repression in the p53 response. Cell 142, 409-419.

*Corresponding author

"We are investigating how lncRNA contributes to the mechanisms that regulate gene expression in cancer cells", Dra. Maite Huarte, Principal Investigator.


Cristina López
Avda. Pío XII, 55
31008 Pamplona

(+34) 948 194 700 Ext. 6021